Address rampant anorectal violence & misinformation!

Blatantly injurious erotic anoreceptive violence—behavior involving a combination of rapid thrusting, considerable girth, and a prolonged duration—is rampant worldwide, facilitated by widespread (willful) ignorance, apathy, and misinformation. It is impossible for resultant anorectal injuries and serious/chronic/permanent health consequences to be uncommon due to anorectal fragility relating to anatomy and neuromuscular physiology [References: Anorectal Risks 1-3]. Hence, when more than one person is involved such violence should constitute a severe crime for any penetrative person. Pornography featuring that violence can have numerous effects on some viewers: inspiring them to emulate what they see, even using coercion; conditioning them to be aroused by suffering; and contributing to development of related mental pathology: sexual sadism disorder, sexual masochism disorder, and perhaps even psychopathy for younger individuals [References: Trends & Associations]. People with those inclinations are having a field day with such violence and spreading disinformation. That unrestrained hedonism is promoting societal decadence. This is happening because the vast majority of humanity likely will never place a high value upon the good health of at least someone else's anus; too many people do not wish to think seriously about anorectal matters even though the anus is one of our most important body parts. Erotic anoreceptive activities therefore must be universally discouraged, and perpetrators of anorectal violence against another person—at least and especially ones who inspire countless others—must face justice by any means necessary. Justice has not been served in far too many cases, and it may be out of governments' hands after too much time has passed. People within governments worldwide who are or were in a position to dispense justice yet failed must be held accountable as well.

Traumatic risks of human anoreceptive activities include inflammation, abrasion and tearing, muscle and connective tissue damage, and colorectal perforation. Sequelae may arise, such as hemorrhage/hematoma, hemorrhoidal disease, ulceration, bacterial infection followed by abscess / fistula / life-threatening systemic sepsis, rectal prolapse, fecal incontinence, anal skin tag (remnant of external hemorrhoidal thrombosis, scar tissue from a healed tear, or a sentinel tag for a chronic anal fissure), and anatomic stenosis (narrowing due to constricting scar tissue). One instance of trauma can lead to multiple complications. Cumulative damage and preexisting conditions are concerns as well. ~2cm beyond the anal opening at the pectinate/dentate line, the epithelium transitions from stratified squamous (anoderm) to a simple columnar mucosal lining in part of the narrow surgical anal canal, continuing along the rectum. That lining is very fragile and easily damaged especially if its mucus barrier is removed by an enema or otherwise impaired. Furthermore, some enemas and lubricants can inflame the lining and even cause it to slough off entirely. Injury to that lining alone elicits no pain sensations due to a lack of somatic innervation, so resultant problems may remain undetected absent one or more obvious symptoms. Neuromuscular physiology also contributes to anorectal fragility particularly for girthy and vigorous insertions, which are objectively foolish and very likely to be significantly injurious. The involuntary internal anal sphincter relaxes with rectal distension. The puborectalis and external sphincter completely relax when a person bears down, causing hemorrhoidal cushions to engorge and become more susceptible to injury by shear (frictional sliding) force.

The human anus when closed at rest should resemble an anteroposterior slit surrounded by shallow, semi-symmetrical, radial perianal skin folds. Healthy perianal skin commonly has a different, typically darker, color due to physiologic hyperpigmentation. Anorectal trauma can result in persistent, externally-visible changes to the anus and its surroundings. Among other outcomes, the anal orifice can become shrunken or misshapen due to constricting scar tissue formation (anatomic anal stenosis), the perianal skin folds may disappear due to underlying anal sphincter damage, and one or more lumps could appear (numerous possibilities, including an anal skin tag, prolapsed internal hemorrhoid, or thrombosed/diseased external hemorrhoid). While the perianal skin folds remain, pruritus and/or cutaneous thickening (i.e., lichenification) may result in exaggerated skin folds. [References for paragraphs 1 and 2: Anatomy & Trauma] A sexual fetish—or at least a strong one—is a requirement for sexual arousal, and a fetishist with partialism obsesses over a (usually non-sexual) body part[1]. A human anus that resembles the anatomical ideal at rest has symmetrical features; symmetry is one aspect that humans consider when evaluating beauty[2]. Partly due to common prejudices against and denigration of the anus along with rampant, anus-mutilating erotic anoreceptive violence, developing an aesthetic anus fetish is highly unusual — and _accepting_ it even moreso. Some malicious people (that is, sexual sadists and sadistic psychopaths) pretend to have such a fetish—and will even praise a clearly damaged/diseased anus typically without being challenged—because doing so facilitates and perpetuates anorectal abuse. 1. "The DSM diagnostic criteria for fetishism." Archives of Sexual Behavior. 2010 Apr; 39(2): 357-62. PMID 19795202. doi:10.1007/s10508-009-9558-7. 2. "Symmetry, beauty and evolution." Nature. 1994 Nov 10; 372(6502): 169-72. PMID 7969448. doi:10.1038/372169a0.

• Strong, repeated shear force in the anal canal is likely to cause permanent damage to supporting tissues of the internal hemorrhoidal cushions at the least, leading to internal hemorrhoidal prolapse (progressively worsening with cumulative damage from repeated trauma). Since healthy internal and external anal cushions help to maintain fecal continence with a watertight seal, anal canal deformation due to their disease or removal can result in fecal incontinence. • Internal rectal prolapse (IRP), aka rectal intussusception, is a common finding among asymptomatic individuals. Strong, repeated shear force in the rectum probably does contribute to development of full-thickness external rectal prolapse (aka procidentia) particularly when IRP is present. Internal hemorrhoidal prolapse—among other conditions—also may contribute to rectal prolapse development. Fecal incontinence can be a consequence of rectal prolapse as well. • Overstretching the anal canal with girthy insertions is likely to cause disruption or fragmentation of one or both anal sphincter muscles, which results in permanent muscle weakening and is associated with fecal incontinence (especially with a damaged or dysfunctional puborectalis muscle). Stretching the anal canal repeatedly with insertions of progressively increasing circumference may cause cumulative muscle damage. • Trauma—including erotic anoreceptive trauma—can instigate development of numerous other anorectal conditions that may cause or lead to fecal incontinence, including fistulas. Surgical treatments for anorectal conditions also can contribute to development of fecal incontinence. [References: Prolapse & Incontinence]